are produced by the same or similar processes, so they tend to show up as a mix. Teasing out the role of any individual element can prove challenging.

A little more than 20 years (SN: 4/6/91, p. 212),
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, studies emerged showing that airborne PM levels below the federal limit were killing people in many U.S. cities. An ambitious hunt immediately commenced to find out how and why. Data on the why are still emerging and a bit equivocal. But clearly,
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, this pollution can damage the lungs,
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, heart and brain.

Ozone,
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, had for years appeared fairly wimpy. Sure, it could aggravate asthma. But for decades there were no data indicating this pollutant would kill people without pre-existing lung disease. And to this day, Devlin notes,
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, data on non-lung impacts from ozone tend to be quite thin.

So until ozone mortality data started to come out around 2004 (SN: 12/11/04, p. 372),
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, his team had little motivation to probe for cardiovascular impacts. But it’s probed them now — and found plenty of potentially adverse changes in a?trial involving 23 healthy young men and women. Some of the more compelling observations involve markers of inflammation, the scientists report in a paper published ahead of print June 25 in Circulation.

Tracy Stevens finds them compelling,
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, anyway. The reason: “The big theory about cardiovascular disease centers

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